Contribution of clumping factor B to pathogenesis of experimental endocarditis due to Staphylococcus aureus

J. M. Entenza, T. J. Foster, D. N. Eidhin, P. Vaudaux, P. Francioli, P. Moreillon

Research output: Contribution to journalArticlepeer-review

Abstract

Staphylococcus aureus Newman with an insertion mutation in clfB, the gene encoding clumping factor B, only marginally decreased infection rate (P > 0.05) in rats with experimental endocarditis. In contrast, clfB complementation on a multicopy plasmid significantly increased infectivity (P < 0.05) over the deleted mutants. Although clfB could affect endovascular infection, its importance in experimental endocarditis was limited.

Original languageEnglish
Pages (from-to)5443-5446
Number of pages4
JournalInfection and Immunity
Volume68
Issue number9
DOIs
Publication statusPublished - 2000
Externally publishedYes

Fingerprint

Dive into the research topics of 'Contribution of clumping factor B to pathogenesis of experimental endocarditis due to Staphylococcus aureus'. Together they form a unique fingerprint.

Cite this